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Importantly, no proliferation of pancreatic acinar cells, such as that seen after management of T3 or perhaps the TRβ agonist GC1 ended up being recognized after either TG68 or IS25, demonstrating the hepato-specificity of these novel TRβ agonists. CONCLUSIONS The current study shows that TG68 and IS25 induce massive hepatocyte proliferation without overt poisoning. Ergo, these representatives may have a substantial medical application for regenerative treatments in liver transplantat
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