https://www.selleckchem.com/pr....oducts/prt062607-p50
Cytochrome c and AIF were also largely increased compared to the interfered group. Moreover, apparent mitochondrial membrane potential loss has also been detected in the CaMK II overexpression group. CONCLUSIONS It suggested that CaMK II induces cell apoptosis. Our findings may give a novel indication that inhibition of CaMK II could be a new way for the therapy of warm ischemia-reperfusion injury after LT in future clinical practice.OBJECTIVE To detect differentially expressed micro ribonucleic acids (miRNAs) in rats with