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A typical attribute on most PRDM genes is always to encode for two primary molecular variants with or with no PR domain. They're produced by either alternative splicing or alternative use of various promoters and play opposite roles, especially in cancer where their imbalance could be usually seen. In this situation, PRDM proteins take part in disease beginning, invasion, and metastasis and their changed expression is related to bad prognosis and clinical outcome. These functions strongly a

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