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001), VWF/ADAMTS-13 (191 219 138;  = 0.003), FVIII (150% 144% 90%;  = 0.001) and TM (5.13 ng/ml 4.91 ng/mL 3.81 ng/ml;   0.001) were only increased in CD regardless of EA status when compared with controls. Lastly, ETP with and without TM remained the same in all three groups. CD patients in clinical remission with EA present endothelial lesion inducing TF exposure and subsequent coagulation cascade activation. Recommended thromboprophylaxis for EA outpatient subgroups will require additional investigation in order to be validated.

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