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In vivo ventricular tachyarrhythmia induction and in vitro optical mapping were utilized to gauge the inducibility of ventricular arrhythmias also to characterize the dynamic properties of electrical prop48±0.04, P0.001). These effects had been strongly connected with down-regulation of KCNN2 (0.38±0.07 vs. 0.74±0.12ng/ml, P0.001). Treating SV additionally decreased the amount of N-terminal pro-B-type natriuretic peptide, cardiac bridging integrator-1, and intramyocardial
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