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Our study provides the first evidence of the potential interaction of BRAF mutation, relaxin, and thyroid hormones in thyroid carcinogenesis. Moreover, our results suggest that hypothyroidism, influenced by RLNH2 activity, may underlie the development of the majority of thyroid cancers and mediate the role of BRAF in thyroid carcinogenesis. BRAF mutation is increasing in papillary thyroid cancers and may be contributing to the rising incidence of this malignancy. Our study provides the first evidence of the potential interaction of BRAF
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