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Extremely, bilateral NA (1 μg/0.2 μl) infusion in to the PFC restored the recognition memory deficits in LC-lesioned rats. These findings suggest that a selective noradrenergic LC lesion induced by 6-OHDA deregulates a noradrenergic system into the PFC, that could be engaged in the early memory impairments observed in nondemented PD patients.Lung disease is considered the most typical and lethal cancerous disease for which the development of efficacious chemotherapeutic agents remains a
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