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l Study, level V.BACKGROUND The aim of the study was to investigate how age and genetic polymorphisms of UGT1A6 and UGT2B7 contribute to the concentrations of valproic acid (VPA) and its hepatotoxic metabolites in Chinese pediatric patients with epilepsy. METHODS A total of 122 children with epilepsy were genotyped at 19TG, 541AG, and 552AC in UGT1A6 and -161CT and 802CT in UGT2B7 using the polymerase chain reaction-restriction fragment length polymorphism method or direct sequencing method. The concentrations of VPA, 4
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