https://www.selleckchem.com/products/ijmjd6.html
MetS may have too complex a mechanistic background to be studied without dissecting the syndrome into its individual (causal) components. Our results indicate that allelic variants of HTR2C genes may have consequences on metabolic parameters. MetS may have too complex a mechanistic background to be studied without dissecting the syndrome into its individual (causal) components.This review discusses the potential mechanistic role of abnormally elevated mitochondrial proton leak and mitochondrial bioenergetic dysfunction in the pathogenesi
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