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In multivariate analysis, reduced 1,5-AG levels were a completely independent predictor of plaque rupture (chances ratio 3.421; P = 0.005) in diabetics with ACS. The area beneath the receiver-operating characteristic bend for 1,5-AG (0.658, P = 0.002) to predict plaque rupture ended up being superior to this for HbA1c (0.587, P = 0.087). Quantities of 1,5-AG were dramatically correlated with urinary 8-iso-prostaglandin F2α amounts (r = - 0.234, P = 0

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