https://plapathway.com/epigene....tic-neuropharmacolog
Consequently, GRASP55 deficiency leads to reduced chylomicron release and uncommonly huge LD formation in abdominal epithelial cells upon exogenous lipid challenge. Particularly, deletion of dGrasp in Drosophila triggers similar defects of lipid accumulation into the midgut. These results highlight the importance of the Golgi complex in cellular lipid legislation, that is evolutionary conserved, and uncover possible therapeutic objectives for obesity-associated diseases.
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