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Pathological discomfort is regulated by a balance between pro-algesic and analgesic components. Communications between opioid peptide-producing immune cells and peripheral sensory neurons expressing opioid receptors represent a strong intrinsic discomfort control in animal designs as well as in humans. Therefore, remedies based on general suppression of immune answers are mostly unsuccessful. It is extremely desirable to build up strategies that specifically promote neuro-immune commu
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