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Unstable functional reentrant circuits known as rotors have been consistently observed in atrial fibrillation and are mechanistically believed critical to the maintenance of the arrhythmia. Recently, using a Poisson renewal theory-based quantitative framework, we have demonstrated that rotor formation (λ ) and destruction rates (λ ) can be measured using in vivo electrophysiologic data. However, the association of λ and λ with clinical, electrical, and structural markers of atrial fibrillation phenotype is unknown. RENEWAL-AF is a multi
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