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More over, optimization associated with CD32+CD4+ T cell purification protocol shows prominent enrichment for HIV DNA (mean, 292-fold) during these cells. Nonetheless, no enrichment for HIV RNA is observed in CD32+CD4+ cells, yielding significantly paid down HIV RNA/DNA ratios. Moreover, HIV proviruses in CD32+CD4+ cells can be reactivated ex vivo to produce virus, highly suggesting why these cells help HIV transcriptional latency. Our outcomes underscore the necessit
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