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Akita mice were highly at risk of AF in colaboration with increased P-wave duration and slowed atrial conduction velocity. In a second model of type 1 DM, mice addressed with streptozotocin (STZ) showed a similar escalation in susceptibility to AF. Chronic insulin treatment paid down susceptibility and period of AF and shortened P-wave length in Akita mice. Atrial activity potential (AP) morphology was modified in Akita mice due to a decrease in upstroke velocity and increases in AP ex
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