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The data also indicate that AG related mito-damage was associated with reduced mitochondrial biogenesis and oxidative phosphorylation, and launch of a greater concentration of mtDNA. Additionally, the info indicated that mtDNA caused histone H3K27 demethylase inhibition, KDM6B, and subsequent swelling by unbalancing mitochondrial redox homeostasis. KDM6B overexpression in AG BMMSCs or AS management in AG mice restores osteogenesis and bone density in vitro as well as in vivo. Mechan
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