https://www.selleckchem.com/products/dbet6.html
5-exposed mice whereas molecules for lipid export were induced after long-term PM2.5 exposure. Therefore, ambient PM2.5 exposure disturbed hepatic lipid metabolism and the effects varied in different exposure duration. These findings in mice provide new insight into the biological basis of PM2.5-induced human metabolic dysfunction and specific strategies may be applied based on different exposure time periods.C60 fullerene (C6 is a nano-pollutant that can damage the respiratory system. Eugenol exhibits significant anti-inflammatory and
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