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Exendin (9-39) antagonised the inhibition of sustenance and water intake and emesis caused by GLP-1 (7-36) amide together with effects on c-Fos expression when you look at the hypothalamus and brainstem, excepting for the bed nucleus regarding the stria terminalis. These data suggest that the action of GLP-1 (7-36) amide to modulate blood glucose, suppress food and water intake and cause emesis include GLP-1 receptors in the hypothalamus and brainstem.This study directed at inv
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