https://www.selleckchem.com/products/cay10444.html
Further investigation revealed that QIG-induced cytotoxicity associated with intracellular ROS accumulation which could suppress the AKT/mTOR signaling pathway, and then induce autophagy and inhibit cell growth. Besides, Erk signaling pathway was also involved in the process of autophagic cell death. Moreover, QIG extract also influenced EMT process and inhibited CRC cell migration. Altogether, this study provides a basis for the utilization of QIG as an alternative medicine for CRC prevention and treatment. Altogether, this study prov
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