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Additionally, tropism towards certain immune-privileged territories may facilitate immune evasion by this virus. Structural homology with SARS-CoV-1 indicates that SARS-CoV-2 can be able to directly impair pRb and p53, that are crucial gatekeepers with tumefaction suppressor features. Furthermore, COVID-19 features preeminent inflammatory response with noticeable oxidative anxiety, which acts as both as initiator and promotor of carcinogenesis. Should there be a carcinogenic threat connected wi
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