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Using electric current density, we identified areas of the dorsal followed by areas of the ventral stream as the source of the re-entrant signal modulating infragranular activity. Our work demonstrates that the VEP does not signal reflect the overall level of a neural response but is the result of an interaction between electric dipoles originating from neural responses in different lamina of V1.The ligand H3 L (6-[3-oxo-3-(2-hydroxyphenyl)propionyl]pyridine-2-carboxylic acid), which exhibits two different coordination pockets, has been e