https://www.selleckchem.com/products/ve-821.html
BACKGROUND The genetic risk associated with rheumatoid arthritis (RA) includes genes regulating DNA methylation, one of the hallmarks of epigenetic re-programing, as well as many T-cell genes, with a strong MHC association, pointing to immunogenetic mechanisms as disease triggers leading to chronicity. The aim of our study was to explore DNA methylation in early, drug-naïve RA patients, towards a better understanding of early events in pathogenesis. RESULT Monocytes, naïve and memory CD4+ T-cells were sorted from 6 healthy controls and 1
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