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This reduced excitability is correlated with faster apical dendrites. Utilizing numerical modelling, we uncover a universal principle underlying the influence of apical length on dendritic backpropagation and excitability, centered on a Na+ channel-dependent broadening of backpropagating action potentials. In conclusion, we provide brand-new ideas to the modulation of dendritic excitability by apical dendrite length and tv show that the working arsenal of L5PNs isn't universal t
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