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We characterized an allele of klp-4 which contains are big indel within the cargo binding domain associated with engine, however, the gene nonetheless is apparently expressed. Behavioral analysis demonstrated that klp-4 mutants have flaws in locomotive signaling, yet not the strikingly uncoordinated movements like those found in unc-104/KIF1A mutants. Animals with this specific large removal are hypersensitive to the acetylcholinesterase inhibitor aldicarb but are unchanged by exogenous serotonin. Interesting