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In addition, the loss of heterozygosity at the TP53 locus was also detected in the dysgerminoma. These results indicated that either the mast cells causing the ASM in this case had originated from the preceding ovarian dysgerminoma as a clonal evolution of a residual tumor cell, which acquired the TP53 mutation, or that both tumors developed from a common cancer stem cell carrying the KIT D816A variation. These results indicated that either the mast cells causing the ASM in this case had originated from the preceding ovarian dysge
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