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Our data show that unlike earlier association of WT1 and 46,XY DSD, ZF4 variations of WT1 are a somewhat typical cause of 46,XX TDSD/OTDSD. This expands the spectrum of phenotypes involving WT1 alternatives and demonstrates that the WT1 protein affecting ZF4 can function as a protestis consider an XX chromosomal context.Protein misfolding and aggregation could be the characteristic of several peoples disorders, including Alzheimer's illness. This process requires the development
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