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The CCS in 30per cent of 108 sporadic adenomas showed p53-upregulated solitary cells, suggesting mounting somatic mutations. No p53-upregulated cells had been found in the crypts of settings. In polypoid adenomas, the mucosa of the stalk without dysplastic tissue over the top disclosed CCS with asymmetrical PC-domains and solitary p53-upregulated cells. The latter observations advised that CCS had developed prior to and never after the development of the dysplastic structure over the top. C
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